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alcoholic encephalopathy mri

Non-alcoholic acute Wernicke's' encephalopathy: role of MRI in non typical cases. PubMed NCBI.
Non-alcoholic acute Wernicke's' encephalopathy: role of MRI in non typical cases. Elefante A 1, Puoti G, Senese R, Coppola C, Russo C, Tortora F, de Divitiis O, Brunetti A. Department of Neuroradiology, University of Naples Federico" II, Naples, Italy. Acute Wernicke's' encephalopathy WE is a severe neurological disorder caused by thiamine deficiency, most commonly found in chronic alcoholics.
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Neuroimaging Findings in Alcohol-Related Encephalopathies: American Journal of Roentgenology: Vol. 195, No. 6 AJR.
In alcoholics with withdrawal seizures, MRI depicts cytotoxic edema during the acute and subacute phases Fig. 8 and significant volume loss in temporal regions 71. It could therefore be deduced that epileptic seizures affect alcoholic subjects similarly to temporal epilepsy, in which reversible edema with some volume loss and consequent hippocampus atrophy is observed. In a patient affected by AWS, reversible vasogenic edema in the cerebellum; thalami; and cortical, subcortical, and deep parietal white matter has been described in the clinical setting of posterior reversible encephalopathy syndrome 72.
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Wernicke Encephalopathy: MR Findings at Clinical Presentation in Twenty-Six Alcoholic and Nonalcoholic Patients American Journal of Neuroradiology.
CONCLUSIONS: Our study confirms the usefulness of MR in reaching a prompt diagnosis of Wernicke encephalopathy to avoid irreversible damage to brain tissue. Contrast enhancement in the mamillary bodies is a typical finding of the disease in the alcoholic population.
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Alcohol-induced changes in the brain as assessed by MRI and CT. PubMed NCBI.
Epub 2009 Dec 8. Alcohol-induced changes in the brain as assessed by MRI and CT. Geibprasert S 1, Gallucci M, Krings T. Division of Neuroradiology, Department of Diagnostic Imaging, University of Toronto, Hospital for Sick Children, Toronto, ON, Canada. This review provides an overview of structural magnetic resonance imaging and computed tomography findings of direct and indirect alcohol-related toxic effects on the brain. In addition to ethanol-related changes to the brain, this article will also describe imaging findings in the acute setting of methanol and ethylene glycol poisoning. Alcohol will lead to brain atrophy, osmotic myelinolysis, Marchiafava-Bignami disease and, especially when related to malnutrition, may also cause Wernicke encephalopathy.
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Neuroimaging of the WernickeKorsakoff Syndrome.
Its relationship with alcoholic intake, feeding habits, organic complications and social problems. Drug Alcohol Depend. 2000; 59 295304. Savage LM, Pitkin SR, Knitowski KM. Rats exposed to acute pyrithiamine-induced thiamine deficiency are more sensitive to the amnestic effects of scopolamine and MK-801: examination of working memory, response selection, and reinforcement contingencies. Behav Brain Res. 1999; 104 1326. Schroth G, Naegele T, Klose U, et al. Reversible brain shrinkage in abstinent alcoholics, measured by MRI. 1988; 30 3859. Schroth G, Wichmann W, Valavanis A. Blood-brain-barrier disruption in acute Wernicke encephalopathy: MR findings.
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WernickeKorsakoff syndrome Wikipedia.
Axial MRI FLAIR image showing hyperintense signal in the periaqueductal gray matter and tectum of the dorsal midbrain. The onset of Wernicke encephalopathy is considered a medical emergency, and thus thiamine administration should be initiated immediately when the disease is suspected.
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Wernicke's' encephalopathy in non-alcoholic patients: A series of 8 cases ScienceDirect.
Encephalopathy was the most frequent onset symptom 4. The classical triad was present in seven patients. Thiamine levels were low in 3/6 and normal in 3/6 cases. MRI was abnormal in seven patients, with high signal intensity in the diencephalon and mammillary bodies 7, periaqueductal grey matter 6, cortex 3 and cerebellum 1.
Wernicke encephalopathy Radiology Reference Article Radiopaedia.org.
Wernicke encephalopathy, also referred as Wernicke-Korsakoff syndrome, is due to thiamine vitamin B 1 deficiency, and is typically seen in alcoholics. On imaging, it is commonly seen on MRI as areas of symmetrical increased T2/FLAIR signal involving the mammillary bodies, dorsomedial thalami, tectal plate, periaqueductal area, and around the third ventricle.

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